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ORDERING INSTRUCTIONS AT THE BOTTOM OF THE LIST BBL No. Antibiotic Agent 231638 Amdinocillin 231596 Amikacin 231597 Amikacin 230701 Ampicillin 231263 Ampicillin 230705 Ampicillin 231264 Ampicillin 231659 Ampicillin with Sulbactam 231660 Ampicillin with Sulbactam 231628 Amoxicillin with Clavulanic Acid 231629 Amoxicillin with Clavulanic Acid 231624 Azlocillin 231625 Azlocillin 230717 Bacitracin 231267 Bacitracin 230721 Bacitracin 231268 Bacitracin 231235 Carbenicillin 231555 Carbenicillin 231652 Cefaclor 231653 Cefaclor 231592 Cefazolin 231593 Cefazolin 231663 Cefixime 231612 Cefoperazone 231613 Cefoperazone 231606 Cefotaxime 231607 Cefotaxime 231655 Cefotetan 231656 Cefotetan 231590 Cefoxitin 231591 Cefoxitin 231632 Ceftazidime 231633 Ceftazidime 231634 Ceftriaxone 231635 Ceftriaxone 231620 Cefuroxime 231621 Cefuroxime 230725 Cephalothin 231271 Cephalothin 230729 Chloramphenicol 230733 Chloramphenicol 231274 Chloramphenicol 231657 Ciprofloxacin 231658 Ciprofloxacin Concentration 10g 30g g 10g 30g units 2 units 10 units 10 units 100g 30g Sale Unit 50 Disc Pack 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs 50 Disc Pack 50 Disc Pack 10 Packs 50 Disc Pack 10 Packs Price USD 18.37 15.77 79.37. Assets, with third parties; Bausch & Lomb's failure to maintain or increase its promotional activity related to RetisertTM; the failure of the ophthalmic medical community in the United States to continue to accept RetisertTM to treat patients with uveitis; issues preventing or delaying the filing of the Company's audited financial statements for the fiscal year ended June 30, 2006, including a reconciliation of the results under US GAAP or other events preventing selling shareholders named in the Company's registration statements to be permitted to utilize those registration statements to sell the securities; the Company's failure to reduce corporate overhead; the Company's failure to successfully move its head office from Perth, Australia to Boston, Massachusetts over the coming months or at all; the failure of MedidurTM for DME to represent a large ophthalmic market opportunity; the failure of any funded development collaborations to result in a fast-to-market solution for our pharma industry partners or a value-generating opportunity for the Company; the failure of our clinical trials for the treatment of steroid-associated elevated intraocular pressure; our failure to obtain required approvals to expand our the Phase IIa trial commenced at Guys & St Thomas' Hospital in London to Singapore; failure of our negotiations with potential licensee parties to share the cost of both the liver and pancreatic cancer trials; our failure to find partners to participate in and fund BioSiliconTM drug delivery R&D programmes; AION's failure to achieve revenues from the BioSiliconTM technology for use in diagnostic products resulting in royalty payments; our inability to achieve milestones and future developments expected to lead to growth of the Company over the coming year; failure of BrachySilTM to represent an effective and user-friendly treatment for pancreatic cancer; our inability to repay the amended convertible notes and new convertible notes; our inability to develop proposed products, including without limitation, in the drug delivery, wound healing, orthopedics, and tissue engineering, diagnostics and food technology fields; failure of our evaluation agreements to result in license agreements; failure to develop applications for BioSiliconTM due to regulatory, scientific or other issues; failure to complete negotiations for new centers for the BrachySilTM Phase IIb clinical trial for inoperable primary liver cancer; failure of our discussions with the FDA for BrachySilTM to continue or to lead to FDA approval; failure of the BrachySilTM Phase IIb clinical trial for inoperable primary liver cancer to determine the optimal dose, provide key safety data or support future pivotal efficacy trials or product registration or approval; failure of the BrachySilTM primary liver program that is in Phase IIb clinical trials to provide a valuable platform for the development and commercialization of BrachySilTM for pancreatic cancer and other indications; failure to commence Phase IIa BrachySilTM trials for the treatment of pancreatic cancer; failure of the findings of the pancreatic cancer Phase IIa trial to provide a platform for further multicenter efficacy and safety trials; failure of there to be optimization and standardization between our two pancreatic cancer study centers; failure of the results of the RetisertTM for DME trial to be a good indicator of the results of pSivida's ongoing Phase III MedidurTM for DME trial; failure of the MedidurTM trials in DME to show a very similar improvement in visual acuity and diabetic retinopathy severity score as RetisertTM for DME; failure of MedidurTM to release fluocinolone acetonide at the same rate as RetisertTM; our inability to recruit patients for the Phase III MedidurTM for DME trial. Other reasons are contained in cautionary statements in the Annual Report on Form 20-F filed with the U.S. Securities and Exchange Commission, including, without limitation, under Item 3.D, "Risk Factors" therein. We do not undertake to update any oral or written forward-looking statements that may be made by or on behalf of pSivida.

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The inclusion of particular patients in clinical trials.8 Research is ongoing to correlate molecular markers in serum or tissue with outcome of disease, and to investigate the use of these biomarkers in RCC prognosis.19 The Von Hippel-Lindau VHL ; gene mutation is seen in a high number 80% ; of RCC cases. Mutations in VHL lead to regulation of downstream genes, including carbonic anhydrase IX CAIX ; . CAIX is induced constitutively in certain tumour types, but is absent in most normal tissues. Staining for the presence of this biomarker has shown to be useful as an independent prognostic marker for RCC.12 Significant numbers of other potentially useful prognostic molecular markers have already been identified from gene array experiments. A prognostic profiling system for metastatic RCC has recently been developed using tissue microarrays. This system was based on eight tumour markers, which had previously been identified as being linked to the development of malignancies. From this study a number of statistically significant independent biomarker predictors for RCC survival were identified.12 Conclusion The variable natural history and lack of systemic therapies for RCC highlight the need for a universally acceptable prognostic model for the disease. This model is likely to result from a consolidation of the historical pathological staging system for RCC, coupled with available new technologies and the use of molecular markers. More accurate prognoses will allow for more exact predictions of patient outcomes from RCC and better management of patients with the disease. There is promise for the development of prognostic tools that will allow more accurate treatment selection and improved surveillance and follow-up after treatment, as well as enhancing clinical trial design, analysis and interpretation.12 48 Crombleholme WR, Schachter J, Ohm-Smith M, Luft J, Whidden R, Sweet RL. Efficacy of single-agent therapy for the treatment of acute pelvic inflammatory disease with ciprofloxacin. American Journal of Medicine 1989; 87 5 A ; : 142S-147S. 49 Martens MG, Faro S, Hammill H, Maccato M, Riddle GD, LaPread E. Comparison of cefotaxime, cefoxitin and clindamycin plus gentamicin in the treatment of uncomplicated and complicated pelvic inflammatory disease. Journal of Antimicrobial Chemotherapy 1990; 26 SUPPL. A ; : 37-43. 50 Buisson P, Mulard C, Baudet J, Bernard P, Mares P, Montero M, et al. [Treatment of upper genital infections in women. Multicenter study of the comparative efficacy and tolerance of an amoxicillinclavulanic acid combination and of a triple antibiotic combination]. [French]. Revue Francaise de Gynecologie et d Obstetrique 1989; 84 10 ; : 699-703. 51 Burchell HJ, Cronje HS, de Wet JI. Efficacy of different antibiotics in the treatment of pelvic inflammatory disease. South African Medical Journal 1987; 72 4 ; : 248-249. 52 Ciraru-Vigneron N, Bercau G, Sauvanet E, Nguyen Tan LR, Felten A, Leaute JB, et al. [The drug combination amoxicillin-clavulanic acid compared to the triple combination in the treatment of severe adnexal infections]. [French]. Pathologie et Biologie 1986; 34 5 Pt 2 ; 665-668. 53 de Beer JA, van den EJ, Odendaal HJ. Efficacy of ampicillin and cefoxitin in the treatment of acute pelvic inflammatory disease. A comparative study. South African Medical Journal 1983; 64 19 ; : 733-736. 54 Judlin P, Koebele A, Zaccabri A, Van Walleghen E, Pavis A, Badonnel Y, et al. [Comparative study of ofloxacin + amoxicillin-clavulanic acid versus doxycycline + amoxicillin-clavulanic acid combination in the treatment of pelvic Chlamydia trachomatis infections]. [French]. Journal de Gynecologie, Obstetrique et Biologie de la Reproduction 1995; 24 3 ; : 253-259. 55 Spence MR, Genadry R, Raffel L. Randomised prospective comparison of ampicillin and doxycycline in the treatment of acute pelvic inflammatory disease in hospitalised patients. Sexually Transmitted Diseases 1981; 8 2 ; : 164-166. 56 Gerstner GJ. [Single administration of 1 g ceftriaxon versus 3 x 1 cefotaxim in the treatment of gynecologic infections--a randomized comparative study]. [German]. Gynakologische Rundschau 1989; 29 3 ; : 182-186. 57 Gerstner GJ. A single dosis of 1 g ceftriaxon and a 3 times 1 g cefotaxim in gynecologic infections compared. A randomised comparative study. Archives of Gynecology & Obstetrics 1989; 245 1-4 ; : 450451. 58 Gerstner GJ. Comparison of ceftriaxone 1 x 1 day ; versus cefotaxime 3 x 1 day ; for gynecologic and obstetric infections: A randomized clinical trial. Gynecologic & Obstetric Investigation 1990; 29 4 ; : 273-277. 59 Gjonnaess H, Dalaker K, Urnes A, Norling B, Kvile G, Mardh PA, et al. Treatment of pelvic inflammatory disease effects of lymecycline and clindamycine. Current Therapeutic Research 1981; 29 6 ; : 885-892. 60 Heinonen PK, Teisala K, Aine R, Miettinen A. Intravenous and oral ciprofloxacin in the treatment of proven pelvic inflammatory disease. American Journal of Medicine 1989; 87 supp 5A ; : 152S-156S. 61 Heinonen PK, Teisala K, Miettinen A, Aine R, Punnonen R, Gronroos P. A comparison of ciprofloxacin with doxycycline plus metronidazole in the treatment of acute pelvic inflammatory disease. Scandinavian Journal of Infectious Diseases - Supplementum 1989; 60: 66-73. Gall SA, Kohan AP, Ayers OM, Hughes CE, Addison WA, Hill GB. Intravenous metronidazole or clindamycin with tobramycin for therapy of pelvic infections. Obstetrics & Gynecology 1981; 57 1 ; : 5158. 63 Ibrahim S, Derde MP, Kaufman L, Clerckx-Braun F, Jacqmin P, Brulein V, et al. Safety, pharmacokinetics and efficacy of once-a-day netilmicin and amikacin versus their conventional schedules in patients suffering from pelvic inflammatory disease. Renal Failure 1990; 12 3 ; : 199-203. 64 Tulkens PM, Clerckx-Braun F, Donnez J, Ibrahim S, Kallay Z, Delmee M, et al. Safety and efficacy of aminoglycosides once-a-day: experimental data and randomized, controlled evaluation in patients suffering from pelvic inflammatory disease. Journal of Drug Development 1988; 1 SUPPL. 3 ; : 71-82. 65 Tulkens PM. Pharmacokinetic and toxicological evaluation of a once-daily regimen versus conventional schedules of netilmicin and amikacin. Journal of Antimicrobial Chemotherapy 1991; 27 Suppl C: 49-61. 66 Loffield RJ, Fijen CA. Antibiotic resistance of Helicobacter pylori: a cross-sectional study in consecutive patients and relation to ethnicity. Clinical Microbiology and Infection 2003; 9 7 ; : 600-604. 67 Drummond MF, O'Brien B, Stoddart GL, Torrance GW. Methods for the economic evaluation of health care programmes. 2nd ed. Oxford: Oxford Medical; 1998. 68 Curran JW. Economic consequences of pelvic inflammatory disease. American Journal of Obstetrics & Gynecology 1980; 138: 848-851.

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Understanding How Strength of Trauma Influences That Memory's Ability to Undergo Reconsolidation Karim Nader Destabilization of Pathogenic Memories in Animal Models of Trauma and Addiction Cristina Alberini Reconsolidation in Humans? Elizabeth Phelps A Novel Treatment for Post-Traumatic Stress Disorder by Reconsolidation Blockade with Propranolol Roger K. Pitman Discussant: Jack Gorman and celestone. Assurance and Didactic Nuclear Medicine for Physicians and Technologists Sponsored by the TechnologistsSection, SNM Northern California Chapter ; 1. Quality Assurance of Scintillation Cameras and Workshop 2. Quality Assurance of Radiopharmaceuticals and Workshop 3. Panel on Laboratory Administrative Problems 15.Any birthdefect, disability, disorder or that mightbe Seeexamples hereditary? considered below ; 16.Any previous with a birthdefect, children disability, geneticdisease? or 17.A brother, sister, parentwith a birthdefect, or disability, geneticdisorder? or 18.Aunts, uncles, nieces, nephews, cousins, grandparents, grandchildren birth or with defects, disabilities, geneticdisorders? or 19.Any familymember with mentalretardation evenmild ; or learning disabilities? 20.Any children who died? and cellcept.

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Manifestations of gingival hyperplasia. Compendium Contin Educ Dent 1990; Supplement 14: S490-S514. 4. Hirtz DG, Nelson KB. The natural hisotry of febrile seizures. Ann Rev Med 1983; 34: 453-71. Millikan CH, ed. Seizure disorders: clinical management. Part II. Detroit: ParkeDavis; 1976: 6-23. 6. Konig SA. Severe hepatotoxicity during valproate therapy: an update and report on eight new fatalities. Epilepsia 1994; 35 5 ; : 1005-15. 7. Harden CL. New antiepileptic drugs. Neurology 1994; 44 5 ; : 787-95. 8. Malamed SF. Managing medical emergencies. JADA 1993; 124 8 ; : 40-51. 9. Selbst SM. Office management of status epilepticus. Pediatr Emerg Care 1991; 7 2 ; : 106-9. 10. Pellock JM. Status epilepticus in children: update and review. J Child Neurol 1994; 9 Supplement 2 ; : S27-35. 11. Rucker LM. Prosthetic treatment for the patient with uncontrolled grand mal epileptic seizures. Spec Care Dentist 1985; 5 ; : 206-7. 12. Braham RL, Casamassimo PS, Nowak AJ, Posnick WR, Steinberg AD. The dental implications of epilepsy. Rockville, Md.: U.S. Department of Health Education and.

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A list of each of the specific drugs that are the subject of the claims herein. This list is attached as Exhibit A to the Complaint. The drugs identified in Exhibit A will be referred to herein as the AWP Inflated Drugs "AWPID" or "AWPIDs" ; . And, in Appendix A, plaintiffs identify the AWP that is the subject of this Complaint for each drug currently at issue pursuant to this Court's Order. Appendix B details which AWPIDs were purchased by each plaintiff.1 and cerezyme. The in vitro interactions between amikacin, netilmicin, tobramycin, gentamicin, and various antipseudomonal P-lactams were studied by the agar dilution checkerboard technique against 30 Pseudomonas aeruginosa strains resistant to all tested antibiotics. Amikacin produced more frequent synergy both at the total and clinically applicable level. Among the P-lactams, clinically relevant synergistic interactions were obtained in the following order: ceftazidime and ceftriaxone moxalactam aztreonam cefotaxime azlocillin cefoperazone cefsulodin carbenicillin.

As the conformational cell binding site P4 and P8 ; . The alternative initiator methionines in the C03 are indicated * ; . The conserved motif VTV, characteristic of all members of the gp85 TS family, and the potential GPI anchor addition consensus sequence are overlined and cerivastatin.

Infecting organism. A K-1-positive, serum-resistant strain of E. coli, originally isolated from a neonate with meningitis, was used. The organism produced , -lactamase, as determined by the nitrocefin chromogenic assay, and was resistant to ampicillin with an MIC and MBC of 32 and 64 , ug ml. The organism was stored on glass beads at -70C, grown for 5 to 6 tryptic soy broth, washed, and diluted in saline to the desired concentration. Antumicrobial agents. Ampicillin and sulbactam were obtained from Roerig Pfizer, New York, N.Y. Ceftriaxone was a commercial preparation Roche Laboratories, Nutley, N.J. ; . Susceptibility tests. MICs and MBCs were measured in Mueller-Hinton broth by the standard macrotube dilution technique, against an inoculum of 5 x iOs CFU ml. The MIC was the lowest concentration that prevented visible growth. The MBC, defined as the concentration that killed .99.9% of the original inoculum, was determined after subculturing 0.1 ml from each clear tube onto a blood agar plate and overnight incubation at 35C. Rabbit model. New Zealand White rabbits weighing 1.8 to 2.6 kg each were anesthetized by intramuscular injection of arepfomazine 3 mg kg of body weight ; , ketamine 30 mg kg ; , and xylazine 15 mg kg ; . The cisterna magna was punctured with a 25-gauge butterfly needle, and the inoculurm of 2 x 105 to 1 x 106 CFU of E. coli in 0.3 ml of saline was administered intracisternally. At 12 to later, the animals were lethargic and febrile. At that time, the animals.

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Aureus infection. For K. pneumoniae infection, the top dosage levels were as follows: ceftazidime, 1 mg kg; ceftizoxime, 0.2 mg kg; cefotiam, 5 mg kg; cefmetazole, 25 mg kg; cefpiramide, 50 mg kg; cefazolin, 25 mg kg; cefuzonam, 5 mg kg; and ceftriaxone, 1 mg kg. Appropriate doses in 0.25 ml of distilled water were given subcutaneously 1 h after challenge. The mice were observed for 4 days. The protective effect of the test drugs was expressed in terms of the 50% effective doses ED50s; in milligrams per kilogram ; calculated by the probit method 4 ; . Therapeutic effect against experimental pneumonia. Male Slc-ICR strain mice age, 4 weeks; weight, 25 to 28 g ; were used in groups of five each. Mice were anesthetized by intravenous injection of 1.5 mg of pentobarbital sodium. K. pneumoniae B-54 that was cultured overnight at 37C on Trypticase soy agar was suspended in physiological saline, and 0.025 ml 5.0 x 105 CFU ; was inoculated intranasally. Doses of 0.4, 2, or 10 mg of ceftriaxone or ceftizoxime per kg or 2 mg of cefuzonam, ceftazidime, cefotiam, or cefpiramide per kg were administered subcutaneously 4, 24, and 36 h after bacterial inoculation. The mice were killed 48 h after challenge, and the lungs were homogenized in physiological saline. Serial 10-fold dilutions of homogenates in saline were prepared, and the numbers of viable organisms in 1 ml the diluted homogenates were measured by the pour plate method. Antibiotic concentrations in sera and lungs. Male Slc-ICR strain mice age, 4 weeks; weight, 25 to 28 g ; were used in groups of 10 each to determine levels in sera and in groups of three each to determine levels in lungs. The antibiotics were given subcutaneously in a single dose of 20 mg kg for the determination of levels in serum and in a single dose of 50 mg kg for the determination of levels in lungs. Blood specimens were taken from the hearts of the mice and serum was separated. The total levels of the test antibiotics in serum were determined by the disk plate diffusion technique by using Bacillus subtilis ATCC 6633 as the test organism for cefazolin, cefotiam, and cefuzonam and Escherichia coli ATCC 39188 for cefmetazole, ceftizoxime, ceftriaxone, cefpiramide, and ceftazidime. Standard solutions for bioassay were prepared with serum. The concentrations of the free fraction in the serum were determined by the ultrafiltration method with a Visking tubing size, 8 32; Wako Chemicals, Ltd., Osaka, Japan ; . After centrifugation at 1, 000 x g for 30 min, the antibiotic concentrations in the ultrafiltrate were determined by the disk diffusion assay. Standard solutions for bioassay were prepared with 67 mM phosphate buffer pH 7.0 ; . Control samples containing 32 or 4 , the test antibiotics per ml in phosphate buffer were handled in the same way to measure the extent of the test antibiotics bound to the Visking tubing. The levels of the test antibiotics in lungs were determined in mice uninfected and infected with K. pneumoniae B-54 24 h after challenge. The mice were killed at specified intervals after drug dosing. The lungs were removed and blotted with filter paper. The lungs of the mice in each group were pooled and homogenized with a Polytron homogenizer after the addition of 2 ml phosphate buffer per g of lung. The homogenates were centrifuged at 10, 000 x g for 10 min. The drug concentration in each supernatant was bioassayed by using a standard curve prepared with the supernatant of lung homogenates of mice uninfected or infected with K. pneumoniae B-54. The hemoglobin levels in supernatants and blood specimens were determined by the cyanmethemoglobin method 15 ; by using Acuglobin Ortho Diagnostic Systems Inc., Raritan, N.J. ; as a standard to estimate the and chamomile.

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J dis child, 1983 nov, 137 11 ; , 1048 - 51 ceftriaxone therapy in pediatric patients ; harrison cj et al; twenty-six children, aged 2 months to 15 years, were treated with intravenous ceftriaxone sodium, 3 5 mg kg every 12 hours, for an average of seven days and ceftriaxone. Of red blood cells from vitamin E-deficient rats and that concurrent lead poisoning accentuated the decrease. Although all and chaparral. SHIPPING: Most orders are shipped within 24 hours via United Parcel Service. FOB origin. Please specify date required on time-sensitive orders. Each order will be charged a handling fee in addition to actual shipping costs plus insurance if valued over 0 ; . Oversize or overweight parcels not deliverable via UPS are shipped via the most convenient, lowest cost carrier servicing your area, FOB origin. Claims for shortage or damaged goods must be made within 5 days after receipt of shipment. EVALUATIONS: Prior arrangements must be made if you wish to order merchandise for evaluation. If not returned within 30 days, we will consider the product sold and payment will be due. Either way, you will be responsible for all freight charges. BACKORDERS: A backorder is any item that is temporarily out of stock, but will be shipped as soon as it is available. Credit card orders will not be charged for backorders until actual shipment. Delays of 4 weeks or longer are unusual. We will notify you if this happens so you have the option to cancel. It is your responsibility to cancel the backorder. You are also responsible for actual shipping charges on backorders. Please advise if your order is to ship complete. SALES TAX: Oklahoma residents are charged appropriate sales tax. Due to the enormous administrative workload created by collecting and reporting sales tax for other states and the thousands of cities we serve, customers in locations outside Oklahoma must be responsible for compliance with their local tax statutes.

Grapefruit juice has been shown to affect the metabolism of several drugs. Included in the list of potential target drugs are diazepam, cisapride, cyclosporine, felodipine and other dihydropyridine calcium channel blockers, midazolam, nisoldipine, triazolam, saquinavir, lovastatin, and atorvastatin. The mechanism of this interaction appears to primarily result from inhibition of enzymes in the intestinal wall. Several constituents of grapefruit juice have been implicated including the flavonoids naringin and naringenin, along with the furanocoumarins, bergapten and 6, 7-dihydroxybergamottin. Unfortunately, the content of these varies between different grapefruit juices and varieties of fruit, making it impossible to determine if one is safer than another and charcoal.

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As expected, sales of rocephin ceftriaxone ; declined markedly following the expiry of the product's us patent in july and the emergence of generic competitors in the second half of the year and celestone. Table 8 incidence % ; of adverse experiences reported during study therapy plus 14-day follow-up in greater than ; 0% of pediatric patients treated with invanz in clinical studies - ticarcillin invanz * + ceftriaxone * clavulanate + adverse events n 384 ; n 100 ; n 24 ; - local: infusion site erythema 9 0 3 infusion site induration 0 0 0 infusion site pain 0 0 2 infusion site phlebitis 8 0 0 infusion site swelling 8 0 2 infusion site warmth 3 0 2 - systemic: abdominal pain 7 0 2 upper abdominal pain 0 0 0 constipation 3 0 0 diarrhea 1 7 1 loose stools 1 0 0 nausea 6 0 0 vomiting 1 2 1 pyrexia 9 0 3 abdominal abscess 0 0 2 herpes simplex 0 0 2 nasopharyngitis 6 0 0 upper respiratory tract infection 3 0 0 viral pharyngitis 0 0 0 hypothermia 6 0 0 dizziness 6 0 0 headache 4 0 0 cough 4 0 0 wheezing 0 0 0 dermatitis 0 0 0 pruritus 6 0 0 diaper dermatitis 7 0 0 rash 9 0 3 - * includes phase iib complicated skin and skin structure infections, community acquired pneumonia and complicated urinary tract infections studies in which patients 3 months to 12 years of age received invanz 15 mg kg iv twice daily up to a maximum of 1 g ceftriaxone 50 mg kg day iv in two divided doses up to a maximum of 2 g, and patients 13 to 17 years of age received invanz 1 g iv daily or ceftriaxone 50 mg kg day iv in a single daily dose and chlorambucil.

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