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Anon., Bangkok Post 12th Jan 1993, "Fishery Cooperatives to Form Federation." Anon., Bangkok Post 16th February 1993, "Fishery Group Seeks National Committee." Anon., Bangkok Post 13th July 1994, "Fishery Team to Visit Thais Held in Burma." Anon., Bangkok Post 13th November 1994, "Burmese Blasted on Fishing." Anon., Bangkok Post 17th November 1994, "VN, Brunei to Join Fish Scheme." Anon., Bangkok Post 31st October 1995, "Venture in Bangladeshi Waters." Anon., Bangkok Post 7th November 1995, "Group to Expand Indonesian Fleet." Anon., Bangkok Post 10th February 1996, "New Office Aims to Revive Dwindling Sea Resources." Anon., Bangkok Post 5th January 1996, "Monthon Issues Sacking Threat in Fisheries Row." Anon., Bangkok Post 9th January 1996, "Monthon Renews War of Words With Fisheries Department." Anon., Bangkok Post 26th March 1996, "Plodprasop Returns for a Third Term at Fisheries." Anon., Bangkok Post 29th July 1998, "Protesters Agree to Lift Blockade." Anon., Bangkok Post 20th April 1999, "B 900m to be Spent on Tuna Fishing Boats From Spain." Apisit Buranakanonda and Woraphan Kanadpon Bangkok Post 26th April 1999, "Thais Warned of Lean Pickings if Plunder Goes on." Apisit Buranakanonda, Bangkok Post 9th February 1999, "Calls for a Regional Approach to Manage Fisheries." Bangkok Post 27th July 1994, "Thailand Seeks Cooperation in ASEAN Joint Fishery Ventures." Bhanravee Tansubhapol, and Achara Ashayagachat, Bangkok Post 6th March 1998, "Push for Completion of Joint Fishing Guide." Phusadee Arunmart, Bangkok Post 3rd November 1995, "Talks to Untangle Knots in Offshore Fishery." Phusadee Arunmas Bangkok Post 19th May 1998, "Taiwanese Urged to Help Fishing Industry." Phusadee Arunmas, Bangkok Post 31st July 1998, "Fishery Pact with Yemen." Phusadee Arunmas, Bangkok Post 20th October 1998, "Foreign Waters Less Friendly in Recession." Phusadee Arunmas, Bangkok Post 31st March 1999, "Thai Fleet is to Fish off Seychelles Coast." Ruj Komonbut, 1995 ; : Thai Fishermen and Their Local Contacts in Irian Jaya; An Assessment of Issues Related to the Spread of HIV AIDS in Merauke. Jakarta, PATH. Smarn Sudto Bangkok Post 26th April 1993, "Panel Approves New Policy to Tackle Fishery Problems." Supang Chantavanit, Paul, Shakti R. and Naing, 1997 ; : Reproductive Health Survey Among Migrant Burmese Women in Ranong Fishing Community. Bangkok. Asian Research Center for Migration, Institute of Asian Studies, Chulalongkorn University. Supradit Kanwanich, Bangkok Post 14th March 1995, "Thai-Burmese Venture Wins B10b Fishing Deal." Supradit Kanwanich, Bangkok Post 15th March 1998a, "Angling for an Agreement." Supradit Kanwanich, Bangkok Post 15th March 1998b, "Conflict on the High Seas." Uamdao Noikorn, Bangkok Post 12th November 1998, "Deep-sea Fishing Promoted to Net More Foreign Exchange." Uamdao Noikorn, Bangkok Post 23rd April 1999, "Fish Stocks Plundered, Experts Warn.
I found out how did they get into the building. b ; * The police discovered who had they beaten up. c ; * I remember clearly how many people did they arrest.
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In general, topical treatments are used to treat mild to moderate, localised psoriasis, whereas systemic therapies are reserved for more widespread or severe forms. Patients with mild disease who do not respond to topical treatment or whose quality of life is severely affected may also be considered for systemic therapy. The latter represents a more aggressive therapy, which is generally effective in the treatment of psoriasis. Combination treatment regimens are used regularly to address the complicated nature of psoriasis management. Rotational therapy, sequential therapy and various combinations of topical and systemic medications have been utilised to enhance efficacy and to minimise the potential adverse effects of any one medication. In clinical decision-making, the choice of treatment is primarily based on evidence of its effectiveness, adverse effects, safety and dis ; comfort. In the Dutch guidelines for photochemotherapy and systemic therapy for chronic severe plaque type psoriasis, the preference for psoriasis treatment is primarily ultraviolet-B UVB ; , followed by photochemotherapy psoralen and UVA PUVA , methotrexate, cyclosporine, acitretin and fumarates. If clearing cannot be achieved by using PUVA, other more aggressive systemic treatments can be used.
Upper Respiratory Medicine, Imperial College School of Medicine at National Heart and Lung Institute, London, United Kingdom; and Department of Allergy and Respiratory Medicine, Guy's Hospital, London, United Kingdom Received for publication September 8, 1999. Accepted for publication June 12, 2000. The costs of publication of this article were defrayed in part by the payment of page charges. This article must therefore be hereby marked advertisement in accordance with 18 U.S.C. Section 1734 solely to indicate this fact.
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Pratt 1996 ; and Britton 1974 ; studied the discrepancies between ICD efficacy studies and concluded there was 40% variation among investigator's ability to reliably define the cause of death. These variations become particularly problematic when the literature is being searched to determine people at highest risk who warrant intervention to reduce their chances of SCD. A classification tool has been proposed to assist with classification of the literature and to encourage the use of more robust definitions in the future ; See Table 16 ; . Table 16: Classification schedule for studies examining sudden cardiac death.
Sequences of both genital and cutaneous HPV types in a small number of keratoacanthomas of nonimmunosuppressed patients. Dermatology. 1999; 198: 122125. Viviano E, Sorce M, Mantegna M. Solitary keratoacanthomas in immunocompetent patients: no detection of papillomavirus DNA by polymerase chain reaction. New Microbiol. 2001; 24: 295-297. Forslund O, DeAngelis PM, Beigi M, Schjolberg AR, Clausen OP. Identification of human papillomavirus in keratoacanthomas. J Cutan Pathol. 2003; 30: 423429. Bravinck JN, Tieben LM, Van der Woude FJ, et al. Prevention of skin cancer and reduction of keratotic skin lesions during acitretin therapy in renal transplant recipients: a double-blind, placebo-controlled study. J Clin Oncol. 1995; 13: 19331938. Kossard S, Artemi P. Acitretin for hypertrophic lichen planuslike reaction in a burn scar. Arch Dermatol. 2000; 136: 591-594 and adalimumab.
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If you are one of the millions of Americans who suffer from kidney stones, you know how painful they can be. Bay Medical Center offers a safe, painless way to end the discomfort--lithotripsy. What are kidney stones? Kidney stones are hard masses that develop from crystals that separate from the urine and build up on the inner surfaces of the kidney. While excruciating pain in the back and side is the hallmark of kidney stones, they may also produce blood in the urine or nausea and vomiting. What is lithotripsy? Extracorporeal shock wave lithotripsy more commonly known as ESWL or lithotripsy ; is a minimally invasive technique that treats kidney stones. Using ultrasound technology, focused shock waves locate and shatter the stone. This procedure is usually performed on an outpatient basis and requires only mild sedation instead of anesthesia. And because the shock waves focus on the stone, the skin and internal organs are not damaged.
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And its progeny, the Second Circuit has noted that: [a]lthough expert testimony should be excluded if it is speculative or conjectural, or if it is based on assumptions that are so unrealistic and contradictory as to suggest bad faith or to be essence an apples and oranges comparison, other contentions that the assumptions are unfounded go to the weight, not the admissibility, of the testimony. Boucher v. United States Suzuki Motor Corp., 73 F.3d 18, 21 2d Cir. 1996 ; internal quotations and citations omitted ; . In determining admissibility under Daubert, trial judges are charged with a gate-keeping function pursuant to Rule 702!
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Traditional agents are primarily administered orally. In one study evaluating the pharmacokinetic pharmacodynamic effects of alefacept, the Psoriasis Area Severity Assessment Index PASI ; and Physician Global Assessment PGA ; were correlated with increasing alefacept serum levels.19 Dose-dependent decreases in peripheral CD4 + memory cells occurred during treatment with alefacept, which was shown to correlate with improvements in psoriasis. It is this specificity for reducing CD4 + memory cells that has been attributed to the sustained efficacy observed with this agent. The pharmacokinetics of etanercept in treating psoriasis are similar to what is observed in patients treated with etanercept for rheumatoid arthritis.20 The pharmacokinetics were similar whether patients were treated with 25 mg twice weekly or 50 mg once weekly, supporting that this dose would be efficacious in treating psoriasis patients. Long-term treatment was also shown to result in similar pharmacokinetics regardless of whether treatment was continuous or intermittent, thus suggesting the potential efficacy of etanercept when used in a sequential or rotational dosing scheme Table 5 ; .21 Methotrexate Methotrexate reduces the synthesis of tetrahydrofolate by binding to dihydrofolate reductase ; and subsequently inhibits pyrimidine synthesis.15 These actions result in a reduction in DNA synthesis, inhibition of mitosis, and a decrease in the proliferation of rapidly dividing cells. Methotrexate is known to decrease T and B cell function and suppress the secretion of cytokines IL-1, interferon-gamma, TNF ; . Acitretin Acitretin is an oral retinoid with anti-inflammatory, antiproliferative, and keratolytic activity.15 Acitretin is the active metabolite of etretinate, previously known as Tegison. While etretinate and agenerase
Disulfide-linked instead to the lysosomally targeted 2-macroglobulin. Consistent with our finding that lysosomes are oxidizing, Feener et al. 44 ; used subcellular fractionation to argue that the disulfide-linked conjugate [125]I-tyramine-SPDP-poly D-lysine ; was not reduced upon delivery to lysosomes. Although our assay may be less sensitive and quantitative than the above radioactive assays, it does offer the ability to image compartments within which reduction and or degradation ; occurs in intact living cells, without any of the potential spatial and temporal artifacts associated with subcellular fractionation. If, as our results suggest, lysosomes are oxidizing, why does this not impede the proteolytic functions of this organelle? Considering the complexity of the lysosomal degradation machinery 45 ; , it may be that proteolytic activity within this compartment is so efficient that reduction of disulfides is not necessary. Alternatively, protein disulfides may be rendered labile within lysosomes despite the oxidative conditions, by analogy to how protein disulfide isomerase within the oxidative ER renders the disulfides of nascent secretory proteins labile to facilitate proper protein folding 46 ; . Whereas disulfide lability within the ER encourages proper nascent protein folding mediated by resident chaperones like BiP [binding protein; 47 ; ], such lability within the acidic and degradative lysosomes would facilitate protein unfolding and proteolysis. Indeed, in antigen-presenting cells, IFN inducible lysosomal thiol reductase GILT ; appears to support processing of at least some antigens 4851 ; . It remains to be determined, however, whether GILT or other oxido-reductases within lysosomes recognize the disulfide SPP or SPDP linkers within antibodydrug conjugates as substrates. Our data suggest that trastuzumab-SPP-RR linker reduction within lysosomes is at best not much more efficient than antibody degradation. If these disulfide linkers are inefficiently reduced within the endocytic pathway, then how can we account for the in vitro and in vivo potency of disulfide-linked antibodyDM1 conjugates such as cantuzumab mertansine [C242-SPP-DM1 12 ; ], anti-PSCA-DM1 52 ; , anti-CD56 huN901-SPP-DM1 53 ; , anti-HER2-SPDP-DM1 8 ; , and trastuzmab-SPP-DM1 20, 21 ; ? Considering that in vitro cytotoxicity studies generally require at least 23 days to detect cell proliferation effects, and in vivo studies require even longer to observe effects on tumor growth, it may be that very slow delivery of free DM1, via inefficient SPP linker reduction, inefficient degradation of the recycling antibody, or both, sufficiently accounts for conjugate potency. Although trastuzumab predominantly recycles, it is slowly degraded within SKBr3 cells with a 1 2.
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Company reports strong growth in revenue and earnings for 2002 Montreal, Canada, February 6, 2003 Paladin Labs Inc. TSX: PLB ; , a Canadian specialty pharmaceutical company focused on marketing and selling urology, endocrinology and women's health products, today announced record financial results for the fourth quarter and year ended December 31, 2002. Highlights of 2002 include: Total revenues increased 31% to .4 million Net income increased to .2 million or ##TEXT##.36 per diluted share Ten new products launched, including Dostinex, Estring, Androderm in a 5 mg format, Rogitine and the Locacorten-Vioform line of products In-licensing of late-stage products from Hydro Med Sciences and Novo Nordisk New Drug Submissions filed with Health Canada for StatexSR and Circadin Application to Health Canada to change Plan BTM from prescription-only to non-prescription status "With record revenue in our fourth quarter, we ended a great year with strong sales momentum, " said Jonathan Ross Goodman, President and CEO of Paladin Labs. "During 2002, we launched ten new products, in-licensed two promising late-stage products, and filed for regulatory approval of two additional brands. We enter 2003 well positioned to continue strengthening our competitive position and our revenue growth potential through an expansion of our sales and marketing activities." Financial Results Revenues for the fourth quarter of 2002 were .2 million, an increase of .3 million or 26%, compared to .9 million in the fourth quarter a year ago. For the year ended December 31, 2002, revenues increased .6 million or 31% to a record .4 million, from .8 million in 2001. Gross profit, as a percentage of revenues, improved to 73% for 2002, compared to 67% in 2001. Net income for the fourth quarter of 2002 was 8, 000 or ##TEXT##.05 per diluted share, compared to a net loss of .5 million or ##TEXT##.12 per diluted share in the same period a year ago. For the year ended December 31, 2002, net income increased to .2 million or ##TEXT##.36 per diluted share from .5 million or ##TEXT##.12 per diluted share for the year ended December 31, 2001. In the fourth quarter of 2002, the Company recorded a 7, 000 charge 6, 000 after tax ; related primarily to the write-down of intellectual property associated with the carrying value of its license for Rogitine. In the fourth quarter of 2001, Paladin recorded a .5 million charge .2 million after tax ; related to the write-down of intellectual property associated with the acquisitions of licenses for ConXnTM relaxin ; , SYNSORB CD, and DepoCytTM and alefacept.
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